|
| Fig. 31.7 Hyperpigmented variant of tinea versicolor. (Courtesy of the Walter Reed Army Medical Center teaching files.) |
In the past,
Malassezi has been suggested to induce hypopigmentation by production of dicarboxylic acid. These compounds do not have a direct effect on melanocytes in tissue culture. The dark lesions of tinea versicolor may be due to a variation in the inflammatory response to the infection. There is not any strong evidence that influences current thought on this question (Fig. 31-7).
Hay RJ, Moore MK: Mycology. In Burns T, Breathnach S, Cox N, Griffiths C, editors:
Rook’s textbook of dermatology, ed 8, vol 2, Malden, MA, 2010, Blackwell Publishing, pp 31.10–31.14.
